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Insulin has been found to activate mTORC1 through the PI3K-Akt pathway [32]

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Elevated glutathione levels protected HepG2 cells not only from oxidative stress (generated by the redox cycler, paraquat) but also from mechanistically diverse stressors such as the inhibition of mitochondrial complex I by rotenone or the inhibition of the proteasomal system by MG-132 and bortezomib (Suppl

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