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Intracellular glutathione levels determine cerebellar granule neuron sensitivity to excitotoxic injury by kainic acid

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When glutathione pathways are disrupted, patients experience a cascade of downstream effects, from mitochondrial damage and severe post-exertional malaise (PEM) to impaired liver detoxification and histamine intolerance

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Defective antigen processing correlates with a low level of intracellular glutathione