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Collectively, the dysfunction of key organellesincluding mitochondria, endoplasmic reticulum, lysosomes, lipid droplets, peroxisomes, and the Golgi apparatussynergistically amplifies ferroptosis susceptibility in COPD

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Research has demonstrated its involvement in wound healing through enhanced collagen and decorin gene expression [1] , activation of tissue remodeling pathways [2] , and regulation of genes associated with antioxidant defense and anti-inflammatory responses [3] [4]

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Steffan JS, Bodai L, Pallos J et al (2001) Histone deacetylase inhibitors arrest polyglutamine-dependent neurodegeneration in Drosophila