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While these strategies remain experimental, they offer hope for patients with recurrent or refractory disease (Plotkin & Blakeley, 2022)

doi: 10.21608/ejh.2021.59941.1425

Their clinical model relies on standard prescriber evaluation and protocol-based dosing

Activation of 1 receptors increases intracellular cAMP, which: Activates protein kinase A (PKA) Phosphorylates L-type calcium channels Increases calcium influx into the cell Calcium entry triggers calcium-induced calcium release (CICR) from the sarcoplasmic reticulum via ryanodine receptors, increasing intracellular calcium available to bind troponin C, thereby enhancing myocardial contractility (positive inotropy)

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Side effects are usually mild and temporary, such as minor bruising, skin spots, or short-lived soreness (International Society for Medical Shockwave Treatment, 2024)