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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

Also, Akt phosphorylates and inactivates pro-apoptotic proteins such as Bad, promoting cell survival and preventing programmed cell death

Theyre everywhere in your gut, your brain, your pituitary gland quietly regulating hunger, fat storage, metabolism and dozens of other processes you never think about

doi: 10.1016/j.ebiom.2016.06.017 82

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Besides positive therapeutic effects, busulfan has some undesirable side effects particularly in cells or tissues with high proliferative activities (38)