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However, further studies have revealed that -cells in the pancreatic islets, and neurons in the nucleus of the solitary tract are also able to produce GLP-1

We demonstrate that the therapeutic potential of GLP-1RAs in RA is mediated by their dual targeting of two key pathological pathways: suppressing a previously unidentified cGAS-STING-ferroptosis axis in chondrocytes and reprogramming synovial macrophages toward an M2 reparative phenotype, which collectively helps maintain joint homeostasis

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Ferdinand, K

At that point, metabolic adaptation is just beginning, lifestyle habits are still forming, and the biological drivers of regain are at their strongest