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Results showed that Endo-FF induced: - Higher levels of ROS in the mouse oocyte - Higher levels of DNA damage in the mouse oocyte - Decrease or a delay in oocyte maturation as compared to controls Oocyte maturation was impaired by Endo-FF and the oocytes development was blocked

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But it is unprecedented for drugs to be put on the pathway to widespread adoption in this manner instead of having to go through the more arduous and evidence-based FDA drug approval process, experts say

TICs maintain lower intracellular ROS levels than other cancer cells within the tumor, primarily through the accumulation of ROS scavengers like glutathione and the overexpression of Gclm and glutathione synthetase, the enzymes necessary for its synthesis, as shown in breast and liver cancer stem cells [18, 20]

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This is especially true in the liver, which has high levels of glutathione