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Although the exact mechanism linking low serotonin levels to migraine pathology has not yet been fully described (Hamel 2007), researchers have hypothesized that serotonin may interfere with pain processing in the brain

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7) and induced ERK-CREB signaling activation, contributing to both Gcg and Vgf upregulation (Supplementary Fig

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Bibcode:2011JExpB.214.3751G

Finally, it is important to acknowledge the similar protective role that UA is proposed to have in Alzheimers disease, indicating an influence not restricted to the dopaminergic neurodegeneration and synucleinopathy in PD