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Similarly, the relevance to disease outcome of molecular remissions seen in some PV patients treated with either IFN- 47,48,50 or busulfan 51 is currently unclear

One plausible explanation for these results is that the gpoA mutant is unable to effectively counteract the elevated ROS concentrations encountered within the intracellular environment of host cells, despite the partial compensatory activity of the TpxD and AhpD peroxidases (Fig

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