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In the CA1 region of the hippocampus, des (13)-IGF-1 increased the field excitatory postsynaptic potentials (fEPSPs), EPSCs, and -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPAR)-mediated postsynaptic exocytosis/endocytosis mechanism [71]
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[68] TEMPOL and similar SOD-mimetic nitroxides exhibit a multiplicity of actions in diseases involving oxidative stress