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It was proposed that GHK-Cu binds to the channels of ferritin involved in iron release and physically prevents the release of Fe (II)

Cellular Pathways GHK-Cu interfaces with signaling pathways such as TGF-, MAPK, and NF-B to influence proliferation and differentiation

Retrieved May 6, 2025, from Burick Center

A 2016 study published in Nature Scientific Reports showed GHK-Cu produced no cytotoxicity to human keratinocytes at concentrations up to 5,800 micromolar over 72 hours and did not induce inflammatory biomarkers unlike other copper compounds

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BPC-157 activates several biological pathways involved in wound repair