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In mouse models genetically predisposed to chronic pain following peripheral injury, DRG-resident macrophages expressing SR-I1 alone (SR-E3 SR-I1 + ) or both SR-E3 and SR-I1 (SR-E3 + SR-I1 + ) were found to promote chronic pain

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Its pervasive natural presence in human plasma, saliva, and tissues has further contributed to its profound relevance in in-vitro laboratory investigations specifically focused on cellular repair, anti-aging mechanisms, and structural adaptation

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