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The electron-withdrawing character and steric bulk of the -amino isobutyric acid residue abolish interactions with the dipeptidyl peptidase 4 active site, rendering the peptide completely refractory to enzymatic degradation [6] [11]

The cortisol elevation is mediated through stimulation of hypothalamic corticotropin-releasing hormone (CRH) release, which in turn activates the pituitary-adrenal axis

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These include neurodegenerative conditions like Parkinsons and Alzheimers, as glutathione protects brain cells from oxidative damage

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