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Under the mediation of p38MAP kinase, heat shock factor 1 (HSF1)-dependent activation of PROM2 transcription can be inhibited by the application of HSF1 inhibitors

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This wide receptor coverage makes it a foundational tool for probing melanocortinreceptor binding, Gproteincoupled cAMP signalling, and downstream transcriptional responses, as well as for comparative selectivity studies against more receptorselective analogues