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By causing hyper-aggregation and hyper-viscosity, which ultimately lead to inflammation and atherogenic events, these results exacerbate endothelial damage (Nabi et al., 2019)

Mitochondria are major contributors to cellular oxidative stress which is acerbated by PQQ to reduce cell death by preserving mitochondrial functions (Hwang and Willoughby, 2018

Also, GHK and GHK-Cu decreased TNF-alpha-dependent secretion of pro-inflammatory IL-6 in normal human dermal fibroblasts NHDF cell line [ 47 ]

Current studies focus on its interaction with signaling pathways involved in inflammatory response and regenerative processes

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