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GLP-1 receptor agonists exert their therapeutic effects through several mechanisms : they stimulate insulin secretion from pancreatic beta cells in a glucose-dependent manner, suppress glucagon release (which reduces hepatic glucose production), slow gastric emptying, and act on appetite centres in the hypothalamus to promote satiety and reduce food intake

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This led to the first approved GLP-1 receptor agonist, exenatide (Byetta), in 2005 which binds to the same receptor but works far longer

While the study did not include follow-up ICP measurements, the reduction in the Frisen grade and retinal nerve fiber layer (RNFL) thickness served as surrogate markers for ICP reduction

This can cause life-threatening breathing problems