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Niacin used to be used quite commonly to boost HDL but trials like AIM-HIGH and HPS2-THRIVE have shown that raising HDL with niacin did not prevent heart attacks
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Current publications present recent data from a critical perspective, specifically from clinical trials in human medicine
Studies have shown that patients suffering from HF tend to have low levels of SCFAs, but elevated levels of TMAO and LPS
the latter, beyond significant gluco-metabolic beneficial effects, have a favorable cardiovascular outcome, reducing cardiovascular events and mortality [32]
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