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The overexpression of glutaminase 2 and SAT1 promotes PUFA oxidation and lipid peroxidation, leading to ferroptosis

As a result, clinicians have avoided using GLP-1 receptor agonists in patients with a history of acute pancreatitis

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found that Exos from hemeified bone marrow MSCs (HO-1/BMSCs) effectively reduced hepatocyte ferroptosis by delivering miR-124-3p, which led to a significant decrease in the iron homeostasis factor, prostate six transmembrane epithelial antigen 3 [168]

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