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In orthopedics models, BPC-157 consistently speeds tendon and muscle healing, with data pointing to angiogenesis modulation (VEGF) during repair

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Abstract Glycogen storage disease type Ib (GSD-Ib), caused by loss-of-function mutations in the endoplasmic reticulum transporter SLC37A4, disrupts glucose homeostasis through impaired glucose-6-phosphate (G6P)/phosphate (Pi) antiport