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It has been shown that pathogenic bacteria or viruses in inflamed tissues promote ferroptosis in cells while activating immune cells and triggering inflammatory responses through the action of lipopolysaccharides, peptidoglycans, bacterial or viral DNA, and host cell toll-like receptors carried or released by themselves (60)

That's a critical distinction

Li, N., et al., Myostatin Attenuation In Vivo Reduces Adiposity, but Activates Adipogenesis

Certain health conditions, physiological states, or expectations may increase risk or limit safety

Intravenous administration results in even more rapid excretion with little opportunity for liver storage

Interestingly, this tamoxifen-induced differentiation occurs in a PKC signaling independent-manner, and not only by activation of classical estrogen receptors but also by activation of GPER-1 (Gonzalez et al., 2016)