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Understanding the specific mechanisms by which DAMPs contribute to these diseases holds significant promise for the development of novel therapeutic interventions

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these focus on the manipulation and administration of several growth factors implicated with healing

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40 In contrast, NO not only antagonizes the effects of AT-II on vascular tone, cell growth and renal sodium excretion but also downregulates the synthesis of ACE and the expression of AT 1 receptors

The mechanism is supportive, not additive