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In the context of ferroptosis, Trx1 supports GPX4 activity and GSH synthesis, and protects mTOR function under oxidative stress, supporting its role as a negative regulator of lipid peroxidation and cell death

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Between 48 and 72 h post-surgery, the binding capacity of 1-AR decreases, which may be associated with reduced 1-AR-mediated signaling for cell proliferation

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