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However, the Glu 138 -Phe 143 sequence clearly has -helical propensity, which is in agreement with the recent crystal structure of the homologous glucagon receptor TM domain showing an extracellular -helical extension of TM1 20

Limitations and challenges Although there has been significant progress in identifying the molecular mechanisms of liver repair and regeneration, there are major difficulties in translating these mechanisms into real clinical applications

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