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Figure 5: Changes in tail motor function in response to treatment with BPC 157 Figure 6: Changes in tail spasticity in response to treatment with BPC 157 The results of the electrophysiology testing suggested that the rats subjected to EMG recordings after surgical spinal cord surgery and treatment with BPC 157, exhibited a significantly lower MUP in the muscles of the tail than was observed in the control group

They found that oA caused neuronal toxicity with lowered DNMT activity, increased APP and PS1 expression, and decreased cell viability, each consequence of which could be prevented by folic acid via methylation-dependent pathways [145]

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