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In addition, to determine how the lack of IGF1R in the lung TME is conditioning tumor growth and metastasis, we performed double fluorescence immunostainings of p-IGF1R with CD68 (TAMs) and FOXP3 (FOXP3 + TILs) in lung sections from LLC-challenged mice

doi: 10.1038/s41598-017-13682-9 159 BabajaniAManzari-TavakoliAJamshidiETarasiRNiknejadH

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