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invaginations, reminiscent of those seen for pU S 3 kinase dead or deletion mutants and some pU L 31 mutants 42,45,58,59,60

It can then cause dysfunction of these brain cells, including increasing microvascular endothelial cells permeability, leucocyte adhesion, procoagulant activity, and reducing the availability of nitric oxide through various biochemical pathways initiated by mitochondrial overproduction of reactive oxygen species (124)

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NAD+ is an essential coenzyme present in every cell within our bodies

Therefore, further studies with this molecule seem to be warranted

There were several study limitations