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Nature 476 (7360), 293297

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However, an additional GPRC6a knockout, involving disruption of the 7-transmembrane and C-terminal region by deletion of exon VI, displays a normal bone phenotype and glucose tolerance (142)

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Autosomal-recessive iron deficiency anemia, dystonia and hypermanganesemia caused by new variant mutation of the manganese transporter gene SLC39A14