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Stress-exposed C- Glud1 +/ mice may model a schizophrenia high-risk population, which is uniquely sensitive to stress-related trigger events

Whether this reflects genuine cardiovascular benefit or simply the healing of minor injuries that were limiting exercise capacity remains unclear

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Note, however, that most of this research was aimed at in-depth analyses of specific forms of homosynaptic plasticity, and experimental conditions were optimized for the induction of these specific plasticity forms, e.g., induction protocols applied at hyperpolarized membrane potentials to maximize calcium influx via CP-AMPARs, and/or recordings were made with cesium-based intracellular solution and added sodium channel blocker QX-314 for better voltage control