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It can be induced by experimental compounds (e.g., erastin, Ras-selective lethal small molecule 3, and buthionine sulfoximine) or clinical drugs (e.g., sulfasalazine, sorafenib, and artesunate) in cancer cells and certain normal cells (e.g., kidney tubule cells, neurons, fibroblasts, and T cells)

To better understand how these processes interact, the hallmarks are often described within a three-tier hierarchy that traces the progression from molecular damage to organismal dysfunction

Gupte, R., Liu, Z

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