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In most cases, it is not understood whether these diseases are caused by the extra chromosome, the altered metabolic profile, or both

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It shows 85% reduction in the -helix with a 30% increase in the -sheet after the synthesis of BSA/AuNCs

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In chemotherapy-sensitive patients, Keap1 (Kelch-like ECH-associated protein 1) binds to NRF2 and mediates NRF2 ubiquitination via the Cul3-E3 ligase complex, promoting its degradation and maintaining NRF2 at low levels [32]