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Scientific inquiries into Mod GRF 1-29 suggest it may engage with receptor sites linked to growth modulation, leading to downstream impacts on protein synthesis and cellular turnover

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Therefore, we present innovative approaches based on targeting on each period of clinical biofilm formation, such as the application of QS inhibitors (QSI) targeting the QS system, drugs that interfere with bacterial metabolic pathways, reagents that can destroy the biofilm components and even combine with activated immune cells such as DCs and macrophages to kill bacteria