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For example, changes in both the presynaptic adhesion molecule neurexins and the postsynaptic adhesion molecule neuroligins have been observed to be associated with changes in synaptic plasticity in early life stress, stressed-sedentary, Parkinsons disease, and Alzheimers disease [54,55,56,57,58]

Therefore, if GLP-1 RA treatment specifically increases the risk of pancreatitis in this vulnerable population, such an effect would likely remain undetected in randomized trials