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Recent lab reports are available in our Lab Reports section

If autoimmunity is present in ME/CFS, it may increase intestinal permeability (518) and explain the various GI manifestations (190)

Consistently, we observe that loss of TBC1D9B, i.e., a genetic condition resembling ARL8B gain-of-function, causes a much less severe lysosomal phenotype than the combined knockout of ARL8A and ARL8B (e.g., in human neurons as reported earlier by us

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Genazzani AD, Santagni S, Rattighieri E, Chierchia E, Despini G, Marini G, et al

Treatment-related adverse events were nearly equal between groups