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FOXA1 can promote the expression of E-cadherin, while BACH1 inhibits the expression of FOXA1 and genes critical for epithelial cell adhesion, including claudin3 and claudin4, thus promoting the EMT

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These results suggest that the upregulation of tribbles homolog 3 protein (TRB3) due to diabetes results in impaired cardiac glucose metabolism via the inhibition of cardiac Akt2 function