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Image: Christoph Burgstedt/Science Photo Library/Getty Images/Getty Images About the Author Howard E

Outside of the canonical GcgR-G s -cAMP axis, other protein interactors and downstream effectors are known to modulate GcgR-mediated signaling and physiological outcomes

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In brain tissue, misfolded proteins such as amyloid-beta and tau can sequester copper ions, creating local copper deficiency while simultaneously generating reactive oxygen species through aberrant metal-protein interactions