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In human studies, GIP promotes lipid disposal by enhancing triglycerides clearance through increased WAT blood flow, increased adipocyte insulin sensitivity, and recruitment of insulin-sensitive LPL, and resultant triglycerides synthesis and storage in WAT [45, 61]

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Research has demonstrated that the accumulation and deposition of MSU crystals in the joint cavity can enhance oxidative stress, releasing large quantities of oxidants such as ROS, nitric oxide (NO) and malondialdehyde (MDA), while suppressing the activity of antioxidants such as superoxide dismutase (SOD) and glutathione (GSH)