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It serves as the primary intracellular thiol antioxidant, participating in detoxification via glutathione S-transferase conjugation, free radical neutralization, immune cell function modulation, and maintenance of other antioxidants (vitamins C and E) in their reduced forms

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69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation

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