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Its mechanism involves specific engagement of AMY1R and AMY3R in hindbrain and hypothalamic circuits that regulate energy homeostasis, leading to preferential fat mass reduction and altered hypothalamic gene expression profiles [2]

(121) found that tripeptides from defatted peanut ( Arachis hypogaea ) meal hydrolysate (DPMH) with Tyr-Gly-Ser (YGS) structure had a strong free radical absorbance capacity (ORAC), preventing linoleic acid peroxidation, and H 2 O 2 induced oxidative damage to PC12 cells had a protective effect