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When released (e.g., with vesicle depletion by reserpine or amphetamine) [69,70,152] it can substitute disturbed dopamine (i.e., shortage (reserpine) [69] or excess (amphetamine) [70]) as competing successfully for sites at dopamine receptors (i.e., counteracting the adverse effects of both dopamine agonists [70,152,153] and dopamine antagonists [68,70,152])

2003 May;5(3):189-94

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This review focuses on the potential of AAV as a gene delivery vector for ovarian cancer therapy, with particular emphasis on approaches involving anti-angiogenic and immune-modulating genes