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Glucose control is the endpoint being managed

However, in genotype 1a replicons, the C316Y substitution has low replication capacity that must be compensated for by additional compensatory substitutions, including L392F or M414T, resulting in an increase in replication levels of at least 10-fold [19]

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Indeed, biochemical evidence has indicated that the TKTL1 protein is not capable of catalyzing a transketolase reaction

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This work was funded by the Deutsche Forschungsgemeinschaft (DFG, German Research Foundation), Project-ID 519777725 to V.L