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doi: 10.1007/s00240-025-01736-x 191 YuKHChenDYChenJHChenSYChenSMChengTTet al
The probable pathway likely involves PCs augmenting GLUT4 translocation and glucose uptake in skeletal muscle through an AMPK-dependent mechanism, subsequently enhancing GLP-1activated insulin signaling and lowering postprandial hyperglycemia
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