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studied the anti-mutagenic effect of low dose (30 nM) selenite-induced GPx activity against -irradiation (8 Gy) by monitoring the mutation frequency of the hprt locus in a panel of four cell lines (NIH3T3, PC12, MCF7, Sup-T1, and AA8) (Table 2) [48, 49]

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placebo, 19) completed the 90-day treatment period

chelation of the copper site by the thiol group, interference with the cellular transfer of tyrosinase to premelanosomes, which is the site of melanin synthesis and indirect inhibition of tyrosinase via its antioxidant effect as the free radical scavenging effect of it blocks the induction of tyrosinase activity caused by peroxides

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