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41 In recent years, research on the differential genes expression and associated pathways in ARDS patient phenotypes has revealed that elevated plasma lactate levels are associated with oxidative phosphorylation and increased gene expression, potentially caused by mitochondrial dysfunction and alterations in energy metabolism 42 (Figure 1)

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Here, we demonstrate for the first time that disruption of gliT , found within the gliotoxin biosynthetic cluster, but subject to differential regulation, completely sensitizes A