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Tericamente, el aumento de la vascularizacin podra alimentar un cncer preexistente , un riesgo potencial que debe ser considerado a pesar de la falta de datos concluyentes en humanos

*Correspondence: C (Linda) MC van Campen, [email protected] This article was submitted to Pharmaceutical Medicine and Outcomes Research, a section of the journal Frontiers in Pharmacology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

The sections were blindly screened, examined for histopathological alterations, and scored by an experienced pathologist

The patient was instructed to not void for two hours

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(2015) reported that the CP-induced tubular damage could be diminished by either knockout or inhibition of RIPK3 or MLKL activity