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The rationale went beyond convenience: vortioxetine acts through multiple serotonergic mechanisms simultaneouslyserotonin transporter (SERT) inhibition alongside 5-HT1A agonism, 5-HT3 and 5-HT7 antagonism, and 5-HT1B partial agonismgiving it a pharmacological breadth suited to a drug-induced depression where the precipitating mechanism was unlikely to be purely serotonergic ( Bupropion XL 150 mg once daily was added specifically to address the dopaminergic dimension

[6] Patients who present clinically with SPCD fall into two categories, a metabolic presentation with hypoglycemia and a cardiac presentation characterized by cardiomyopathy

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