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CPT1A, with its full-length cDNA clone isolated from rat liver that predicted a protein of 773 amino acids (van der Leij et al., 2000) and has lower affinity for substrate carnitine (McGarry and Brown, 1997)

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ERAD helps mitigate this burden by degrading these proteins, thereby preventing their aggregation and the subsequent activation of detrimental pathways, such as chronic ER stress, autophagy dysfunction, and apoptotic signaling