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The key takeaway is that GHK-CU produces measurable, documented improvements when used consistently at appropriate doses

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[115] This functionality is lost in vitamin B 12 deficiency, resulting in an increased homocysteine level and the trapping of folate as 5-methyl-tetrahydrofolate, from which THF (the active form of folate) cannot be recovered

Some research has explored whether AHK-Cu may help modulate 5-alpha reductase, the enzyme that converts testosterone to DHT