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288 In contrast, in subarachnoid hemorrhage models, ACSS2 activation upregulates ATG5 expression and enhances autophagy, reducing neuronal apoptosis and brain edema

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The process typically begins within hours of administration and can remain active for 12-24 hours based on the sustained activation of lipolytic enzymes and metabolic pathways

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Hier werden vermehrt Sauerstoffradikale bei der Zusammensetzung der Schilddrsenhormone freigesetzt