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In addition to targeted and untargeted metabolomics in RA, integrating proteomics, transcriptomics, and genomics can help reveal the specific disease pathogenesis (68)

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RAGE knockout models provide mechanistic support for the receptor-level contribution of RAGE signaling to inflammatory tissue injury, although direct evidence in human varicocele-associated ED remains limited ( Sympathetic nervous system overactivity and catecholamine release may further aggravate endothelial and vascular dysfunction (127)

It is mediated by iron overload, which results in reactive oxygen species (ROS) accumulation, glutathione depletion, lipid peroxidation, and ultimately cell death [7]