Glutathione Depletion Causes Glutathione synthetase deficiency People with this rare genetic disorder do not have enough of the glutathione synthetase molecule to produce glutathione
In vivo administration significantly increases serum testosterone levels in aged mice, accompanied by elevated expression of key testosterone synthesis enzymes including 3-HSD and CYP11A1
Calcium influx in response to glutamate puffs at one dendritic location was mediated almost exclusively by mGluRs with negligible contribution of NMDARs, AMPARs, and VGCCs while, at another location on the same cell, calcium responses were mediated by NMDARs, AMPARs, and VGCCs, and sequential blockade of these sources gradually reduced and eventually eliminated calcium responses (Topolnik et al., 2005)
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This post-translational modification is now recognised as a major mechanism of redox-regulated cell signalling, controlling the activity of transcription factors, kinases, phosphatases, and metabolic enzymes in response to the intracellular redox environment
